Full-length and N-terminally truncated recombinant interleukin-38 variants are similarly inefficient in antagonizing interleukin-36 and interleukin-1 receptors - revised version

This submission contains the primary datasets for all Figures and Supplementary Figures of the revised version of the manuscript.

Organizational unit: Group 1022 Palmer

Type: Dataset

DOI: 10.26037/yareta:btrvdygarvahzcyvhq6h7e56me

License: Creative Commons Attribution 4.0 International

Publication date

11/12/2024

Retention date11/10/2034

Access levelPublic

SensitivityBlue

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Contributors

Palmer-Lourenco, Gaby
Diaz Barreiro, Alejandro

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118 Files (3.7 GB)

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Group 1022 Palmer

Oxidation-sensitive cysteines drive IL-38 amyloid formation

2024

Public 9.6 GB

Group 1022 Palmer

Oxidation-sensitive cysteines drive IL-38 amyloid formation (revisions)

2024

Public 60.6 MB

Group 1022 Palmer

Full-length and N-terminally truncated interleukin-38 variants are similarly inefficient in antagonizing interleukin-36 and interleukin-1 receptors

2024

Public 2.5 GB

Group 1022 Palmer

Full-length and N-terminally truncated recombinant interleukin-38 variants are similarly inefficient in antagonizing interleukin-36 and interleukin-1 receptors

2024

Public 1.1 GB

Full-length and N-terminally truncated recombinant interleukin-38 variants are similarly inefficient in antagonizing interleukin-36 and interleukin-1 receptors - revised version

This submission contains the primary datasets for all Figures and Supplementary Figures of the revised version of the manuscript.

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